Foggy Oldie
New User
Hi everyone.
Just wondering if anyone has had an MRI report with similar findings? I posted a while ago about our pony who is still rehabbing following a bilateral navicular bursoscopy off the back of this report. She has had steroid injections in front feet and hocks. Most recently (just over a week ago) she has had Arthramid injections in her fronts due to becoming lame again. The vet mentioned arthritis on our last visit which I hadn’t picked up on in the report but I’m guessing he’s referring to the osteophyte formations in point 2?
1- Deep digital flexor tendon- there is damage to the dorsal surface of the DDFT in both front feet. In the medial lobe in the left front foot the is some hyperintense signal from the dorsal aspect of the tendon in T1 and T2 weighted sequences with a soft tissue mass on the surface of the tendon. Additionally there is soft tissue mass on the surface of the lateral lobe and in both the left fore and right fore. There is moderate to marked effusion of the navicular bursa in both feet and this is causing a dorsal deviation and thinning of the navicular collateral ligament at the proximal recess of the bursa. The damage to the tendon does not appear to propagate in to the Pastern region or further distally towards the insertion and appears to be isolated to the suprasesamodian region.
2- proximal interphalangeal joint- there is mild osteophyte formation at the margins of the joint in both front feet.
3- pedal bone- there is mild, diffuse increase in STIR signal throughout the pedal bone in the right front foot.
Comments- The most significant damage is the damage to the DDFT and this is likely the cause of lameness in this case. The damage appears to be isolated to the surface and superficial dorsal surface in both feet., but there is extrusion fibres into the bursa in both feet. It is difficult to be sure about the chronicity of the injury. The fluid signal in the tendon injury and the bursitis suggest active inflammatory change but I suspect that the surface damage is more long-standing. This presents a balance in treatment options between anti-inflammatory and regenerative options. The lesions would also be visible surgically, via navicular bursoscopy.
1- Deep digital flexor tendon- there is damage to the dorsal surface of the DDFT in both front feet. In the medial lobe in the left front foot the is some hyperintense signal from the dorsal aspect of the tendon in T1 and T2 weighted sequences with a soft tissue mass on the surface of the tendon. Additionally there is soft tissue mass on the surface of the lateral lobe and in both the left fore and right fore. There is moderate to marked effusion of the navicular bursa in both feet and this is causing a dorsal deviation and thinning of the navicular collateral ligament at the proximal recess of the bursa. The damage to the tendon does not appear to propagate in to the Pastern region or further distally towards the insertion and appears to be isolated to the suprasesamodian region.
2- proximal interphalangeal joint- there is mild osteophyte formation at the margins of the joint in both front feet.
3- pedal bone- there is mild, diffuse increase in STIR signal throughout the pedal bone in the right front foot.
Comments- The most significant damage is the damage to the DDFT and this is likely the cause of lameness in this case. The damage appears to be isolated to the surface and superficial dorsal surface in both feet., but there is extrusion fibres into the bursa in both feet. It is difficult to be sure about the chronicity of the injury. The fluid signal in the tendon injury and the bursitis suggest active inflammatory change but I suspect that the surface damage is more long-standing. This presents a balance in treatment options between anti-inflammatory and regenerative options. The lesions would also be visible surgically, via navicular bursoscopy. Any comments gratefully received.
Just wondering if anyone has had an MRI report with similar findings? I posted a while ago about our pony who is still rehabbing following a bilateral navicular bursoscopy off the back of this report. She has had steroid injections in front feet and hocks. Most recently (just over a week ago) she has had Arthramid injections in her fronts due to becoming lame again. The vet mentioned arthritis on our last visit which I hadn’t picked up on in the report but I’m guessing he’s referring to the osteophyte formations in point 2?
1- Deep digital flexor tendon- there is damage to the dorsal surface of the DDFT in both front feet. In the medial lobe in the left front foot the is some hyperintense signal from the dorsal aspect of the tendon in T1 and T2 weighted sequences with a soft tissue mass on the surface of the tendon. Additionally there is soft tissue mass on the surface of the lateral lobe and in both the left fore and right fore. There is moderate to marked effusion of the navicular bursa in both feet and this is causing a dorsal deviation and thinning of the navicular collateral ligament at the proximal recess of the bursa. The damage to the tendon does not appear to propagate in to the Pastern region or further distally towards the insertion and appears to be isolated to the suprasesamodian region.
2- proximal interphalangeal joint- there is mild osteophyte formation at the margins of the joint in both front feet.
3- pedal bone- there is mild, diffuse increase in STIR signal throughout the pedal bone in the right front foot.
Comments- The most significant damage is the damage to the DDFT and this is likely the cause of lameness in this case. The damage appears to be isolated to the surface and superficial dorsal surface in both feet., but there is extrusion fibres into the bursa in both feet. It is difficult to be sure about the chronicity of the injury. The fluid signal in the tendon injury and the bursitis suggest active inflammatory change but I suspect that the surface damage is more long-standing. This presents a balance in treatment options between anti-inflammatory and regenerative options. The lesions would also be visible surgically, via navicular bursoscopy.
1- Deep digital flexor tendon- there is damage to the dorsal surface of the DDFT in both front feet. In the medial lobe in the left front foot the is some hyperintense signal from the dorsal aspect of the tendon in T1 and T2 weighted sequences with a soft tissue mass on the surface of the tendon. Additionally there is soft tissue mass on the surface of the lateral lobe and in both the left fore and right fore. There is moderate to marked effusion of the navicular bursa in both feet and this is causing a dorsal deviation and thinning of the navicular collateral ligament at the proximal recess of the bursa. The damage to the tendon does not appear to propagate in to the Pastern region or further distally towards the insertion and appears to be isolated to the suprasesamodian region.
2- proximal interphalangeal joint- there is mild osteophyte formation at the margins of the joint in both front feet.
3- pedal bone- there is mild, diffuse increase in STIR signal throughout the pedal bone in the right front foot.
Comments- The most significant damage is the damage to the DDFT and this is likely the cause of lameness in this case. The damage appears to be isolated to the surface and superficial dorsal surface in both feet., but there is extrusion fibres into the bursa in both feet. It is difficult to be sure about the chronicity of the injury. The fluid signal in the tendon injury and the bursitis suggest active inflammatory change but I suspect that the surface damage is more long-standing. This presents a balance in treatment options between anti-inflammatory and regenerative options. The lesions would also be visible surgically, via navicular bursoscopy. Any comments gratefully received.
